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Ipamorelin
& CJC-1295

Ipamorelin: NNC 26-0161 · CJC-1295: DAC:GRF · Drug Affinity Complex

"The most popular GH secretagogue stack in existence. CJC-1295 sustains the baseline. Ipamorelin fires the pulse. Together they mimic youthful GH patterns more completely than either alone — the tortoise and the hare as relay partners."

Ipamorelin
Ghrelin receptor agonist · 5 aa
CJC-1295
GHRH analogue · DAC modified
Status
UK: not illegal to buy or possess · WADA: prohibited at all times · US FDA: not approved · research compounds
Half-lives
Ipa: ~2h · CJC: 6–8 days (DAC)
Protocol summary
Ipamorelin
200–300 mcg per dose
CJC-1295 no-DAC
100 mcg per dose · 2–3× daily SubQ
Cycle
8–16 weeks, then 4 wks off
Community-reported
Ipamorelin 200–300 mcg + CJC no-DAC 100 mcg · SubQ · 2–3× daily fasted
Inject fasted; insulin suppresses GH release
How we read the evidence
The most-used GH secretagogue stack · synergistic GHRH + ghrelin receptor activation · cleanest side-effect profile of the GHRP class · cycling required
Animal evidence

Synergistic mechanism well-characterised. Ipamorelin (pentapeptide) selectively agonises the ghrelin receptor (GHS-R1a) on pituitary somatotrophs, triggering rapid GH pulses without significantly affecting cortisol, ACTH, or prolactin — distinguishing it from older GHRPs (GHRP-2, GHRP-6, hexarelin) which all carry meaningful cortisol/prolactin spillover. CJC-1295 no-DAC (also called Mod-GRF 1-29) is a GHRH analog stabilised to ~30 minute half-life — paired with ipamorelin, the two activate complementary receptor systems and produce a GH pulse significantly larger than either alone (Alba et al. 2006; multiple subsequent studies).

Community & clinical practice

Community protocols converge tightly on 200–300 mcg ipamorelin + 100 mcg CJC-1295 no-DAC SubQ, 2–3× daily on empty stomach, with bedtime dose being most important (synergises with the natural nocturnal GH surge). Some clinical protocols escalate gradually (0.6 mg total daily in week 1 increasing to 2.0 mg by month two). Cycle 8–16 weeks then break 4 weeks. With Ipamorelin's selectivity, the side-effect profile is the cleanest in the GHRP class — minimal cortisol, no significant prolactin elevation, mild appetite increase compared to GHRP-6. Common side effects: water retention (most pronounced weeks 1–3 then adapts), mild hunger, injection-site reactions. Cycling is required — chronic use produces GHS-R1a downregulation and the response stops.

Human trial data

Foundational pharmacokinetic work in Journal of Clinical Endocrinology and Metabolism (Teichman et al. 2006 for CJC-1295, separate Phase 1 work for ipamorelin). Surgical proof-of-concept trials used weight-based dosing (e.g. 0.03 mg/kg twice daily). Clinical evidence at the stack level (rather than individual compounds) is community-derived rather than RCT — no large Phase 3 stack trial exists. Hormonal effects (GH and IGF-1 elevation) are well-characterised; aesthetic and body-composition outcomes are less rigorously studied. Marketing claims of dramatic short-term transformations exceed what trial-level evidence supports.

Regulatory status

Neither compound is FDA-approved for therapeutic use. Both available via compounding pharmacies and research vendors. WADA-banned for tested athletes. The CJC-1295 Phase 2 halt in 2006 (HIV lipodystrophy trial discontinued after a participant death attributed to pre-existing cardiovascular disease) is part of the honest evidence picture — pharma development stopped, no further FDA pathway pursued. Long-term safety beyond Phase 1 timeframes is essentially uncharted.

Convergence

The ipamorelin + CJC-1295 no-DAC stack is the standard modern GH secretagogue protocol — synergistic, well-tolerated, with the cleanest side-effect profile in the class. Standard protocol: 200–300 mcg ipamorelin + 100 mcg CJC-1295 no-DAC SubQ 2–3× daily, 8–16 weeks then 4 weeks off. Hormonal effects (GH elevation, IGF-1 elevation) are real and measurable; aesthetic results are gradual (weeks to months) and depend on training and nutrition. Pep IQ flags this honestly: cycling is non-negotiable, marketing claims of dramatic transformation exceed evidence, and the long-term safety profile is uncharted but appears favourable in short-cycle use. The cleanest entry into GH-secretagogue protocols if cycled responsibly.

Origin & Background

Pulse & sustain — the GH stack

Ipamorelin was first described in a landmark 1998 paper by Raun et al. as "the first selective growth hormone secretagogue" — notable because it stimulated GH release without the cortisol, prolactin, or ACTH elevations seen with earlier GH secretagogues like GHRP-2 and GHRP-6. That selectivity made it uniquely clean for body composition applications.

CJC-1295 is a GHRH analogue with a Drug Affinity Complex (DAC) modification — a chemical anchor that binds to albumin in the bloodstream and dramatically extends its half-life from minutes to 6–8 days. A 2006 Phase II trial in healthy adults by Teichman et al. (published in JCEM) showed that a single dose produced sustained, dose-dependent increases in GH and IGF-1 lasting up to 28 days — a landmark result for a GHRH analogue.

Neither compound is FDA-approved as a drug. They are available through compounding pharmacies and are among the most prescribed peptides at anti-ageing and functional medicine clinics globally. The combination emerged from the logic that CJC-1295 sustains the GHRH signal while ipamorelin amplifies individual GH pulses — covering two complementary aspects of GH physiology simultaneously.

The "pulse and sustain" mechanism: CJC-1295 maintains an elevated GHRH baseline over days, priming the pituitary. Ipamorelin then triggers sharp GH pulses through a completely different receptor (GHSR — the ghrelin receptor). Studies suggest the combination produces 3–5x more GH release than either compound alone, because they activate two distinct pathways simultaneously.

Science & Mechanism

Two peptides, two receptors

How They Work Together

1
CJC-1295 — GHRHR pathway: Binds to GHRH receptors on pituitary somatotrophs. The DAC modification binds albumin, creating a depot that continuously releases active peptide over 6–8 days. This "raises the floor" of GH secretion — maintaining steady elevated baseline GH and IGF-1.
2
Ipamorelin — GHSR pathway: Binds to the ghrelin receptor (growth hormone secretagogue receptor) on pituitary cells. Acts independently of the GHRH pathway. Triggers rapid, sharp GH pulses — the "spike" on top of CJC-1295's elevated baseline. Half-life ~2 hours; effect peaks within 30–60 minutes.
3
Selectivity — why ipamorelin is different: Raun's 1998 study established that ipamorelin does not stimulate cortisol, ACTH, prolactin, or aldosterone at therapeutic doses — making it the cleanest GH secretagogue known. GHRP-2 and GHRP-6 stimulate cortisol and hunger; ipamorelin does not.
4
IGF-1 downstream: Combined GH elevation from both pathways drives hepatic IGF-1 production. IGF-1 mediates the anabolic, lipolytic, and regenerative effects. The combination's 3–5x GH amplification translates to correspondingly greater IGF-1 elevation.
5
Feedback preservation: Both compounds work through the pituitary's natural mechanisms. Somatostatin feedback still operates, providing a regulatory ceiling. This is physiologically safer than direct GH injection but the ceiling is higher and more sustained than natural secretion.
Benefits & Evidence

What the data shows

💪
Lean muscle mass and body composition
GH and IGF-1 increase protein synthesis and nitrogen retention. Most users report significant body composition improvements over 3–6 months when combined with resistance training. The IGF-1 elevation from the combination is substantially greater than from either compound alone.
● Moderate — extrapolated from GH/IGF-1 data
🔥
Fat loss — particularly visceral
GH has direct lipolytic effects, mobilising stored fat for energy. CJC-1295 Phase II data (2006) showed significant increases in mean GH and IGF-1 associated with improved metabolic markers. Visceral fat is particularly responsive to GH-axis restoration.
● Moderate — CJC Phase II trial evidence
💤
Sleep quality and recovery
GH secretion is tightly coupled to slow-wave sleep. Raising GH signalling deepens slow-wave sleep in a positive feedback loop. Recovery from training accelerates significantly — typically the first noticeable effect.
● Moderate — consistent across reports
🩹
Tissue repair and injury recovery
IGF-1 drives satellite cell activation and protein synthesis in muscle and connective tissue. Evidence is mixed — a 2020 pilot study showed GH secretagogues preserved muscle strength after ACL reconstruction; a 2024 in vitro study showed no direct tendon cell effect. The systemic IGF-1 elevation likely matters more than any direct GH effect.
● Limited — mixed evidence
Energy, cognition and anti-ageing
GH insufficiency produces fatigue, cognitive fog and reduced wellbeing. Restoring GH/IGF-1 toward younger levels addresses these symptoms. Most consistent in adults over 40 with measurable GH decline.
● Moderate — consistent with GH restoration
Things to know

Risks & considerations

⚠️
Use with medical supervision. Neither compound is FDA-approved. The elevated IGF-1 produced by this combination is substantially greater than sermorelin alone and requires regular monitoring. Long-term safety data is limited — clinical use is based on extrapolation from GH physiology rather than direct long-term trials of this specific combination.
Mild
Water retention — the most common complaint. IGF-1-mediated sodium retention causes bloating and joint puffiness. Dose-dependent and reversible. Usually resolves by reducing dose.
Mild
Injection site reactions — transient redness or discomfort. Common with SubQ administration. Rotate injection sites.
Mild
Headache and flushing — typically in the first few weeks. Usually self-resolving.
Moderate
Elevated IGF-1 — sustained high IGF-1 carries theoretical cancer-promotion risks (IGF-1 is a general growth signal). Regular monitoring and keeping IGF-1 within age-appropriate reference ranges is essential.
Moderate
Insulin sensitivity changes — GH at elevated levels promotes insulin resistance. Monitor fasting glucose and HbA1c during extended protocols.
Serious
Contraindicated in active malignancy — GH axis stimulation is contraindicated in anyone with active cancer or without appropriate oncological clearance.

⚠ Key Warnings

Monitor IGF-1, fasting glucose and HbA1c every 3 months during use. Do not allow IGF-1 to exceed the upper limit of the age-appropriate reference range.
WADA prohibited at all times for all competitive athletes under anti-doping programmes.
CJC-1295 with DAC produces prolonged GH elevation — this cannot be rapidly reversed if side effects appear. Some practitioners prefer CJC-1295 without DAC (Mod-GRF 1-29) for more controllable shorter-acting effects.
These are research compounds. Quality and dosing accuracy vary significantly between compounding pharmacies — use PCAB-accredited facilities.